A vein in the umbilical cord; returns nutrient blood from the placenta to the fetus.
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Primary cultured human umbilical vein endothelial cells were used in this study.
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We studied their direct antiangiogenic effects on human umbilical vein endothelial cells.
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Tumor conditioned medium induced proliferation of human umbilical vein endothelial cells.
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This was also observed in situ in the endothelial cells in the umbilical vein.
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The expression of CD105 decreased in sclerosant treated cultured human umbilical vein endothelial cells.
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Additionally, the in vitro studies were performed with use of human umbilical vein endothelial cells.
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Recombinant human CTGF could enhance proliferation and Transwell migration of human umbilical vein endothelial cells.
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In human umbilical vein endothelial cells, this occurred at what are likely physiologic intracellular ascorbate concentrations.
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The umbilical artery is slightly more sensitive to the effects of estradiol than the umbilical vein.
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SVF cells were found to work together with human umbilical vein endothelial cells to form capillary networks.
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Here, human umbilical vein endothelial cells were engineered using adenovirus vectors to express either ERalpha or ERbeta.
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Inhibition of PGHS activity by acetaminophen in human umbilical vein endothelial cells is abrogated by t-butyl hydroperoxide.
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Methods and results: Sera from healthy children and KD subjects were incubated with human umbilical vein endothelial cells.
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After separation, the anti-inflammatory effects of the two compounds were evaluated using lipopolysaccharide-induced human umbilical vein endothelial cells.
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Cell proliferation assay showed that HRP-3 stimulated the growth of human umbilical vein endothelial cells (HUVECs).
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We have shown that these peptides inhibit proliferation and migration of human umbilical vein endothelial cells in vitro.
