Tih2p is required for vegetative cell growth and localizes in the nucleus.

By contrast, in the holo-bLf model, only a modest vegetative cell population was observed until 16 days post antibiotic administration.

In separate batch culture experiments, holo-bLf prevented C. difficile vegetative cell growth and toxin production, whereas apo-bLf and iron alone did not.

We showed that all 13 myosins were expressed in vegetative cells.

C. difficile infections begin when its metabolically dormant spores germinate to form toxin-producing vegetative cells.

At - 8 °C, the young vegetative cells were unable to recover from severe frost damage.

These results indicate that young vegetative cells can adapt better to the experimental UV-B stress than pre-akinetes.

Upon entering the gut, C. difficile spores germinate and outgrow to produce vegetative cells that release disease-causing toxins.

That, however, does not prevent them from differentiating into animal and vegetative cells, even during the cleavage.)

The degree of photoinhibition was higher in spores than in vegetative cells, indicating that spores are less susceptible to photodamage.

A microscopic picture of spores and vegetative cells of Bacillus anthracis which causes the disease anthrax is pictured in this undated file photograph.

However, upon sensing specific bile salts in the vertebrate gut, C. difficile spores transform back into the vegetative cells that are responsible for causing disease.

The gene became deregulated when integrated at nonhomologous chromosomal sites, in that transcript levels were elevated in vegetative cells (hyphae) and variably altered in conidia.

G alpha 1 is expressed in vegetative cells through aggregate stages while G alpha 2 is inducible by cAMP pulses and preferentially expressed in aggregation.