These infusion rates produce plasma levels similar to those seen during corticotropin stimulation.
2
For example, corticotropin-releasing factor regulates the homeostatic pattern of visceral functions during stress.
3
Concentrations of endorphins and corticotropin increased significantly also during the anaerobic exercise test.
4
A Sheehan event probably occurred 33 years before the patient presented with corticotropin deficiency.
5
Thirty-one patients underwent also a human corticotropin releasing hormone test.
1
Corticotropin releasing factor and adrenocorticotropin stimulation tests, as well as 24-hour urinary-free cortisol measurements were performed.
2
When the cells are stimulated by the secretagogue 8-bromo-cAMP, these polypeptides are coordinately secreted with mature adrenocorticotropin into the culture medium.
3
In neither case was the sorting or secretion of adrenocorticotropin detectably altered, suggesting that the proteoglycan is not required for these processes.
4
The AtT-20 cell, a mouse pituitary tumor line that secretes adrenocorticotropin and beta-endorphin, sorts the proteins it externalizes into two exocytotic pathways.
5
Subsequently, 50% of the birds from each dosage group were subjected to a 6-day adrenal function test, whereby they were injected with long-lasting adrenocorticotropin (ACTH).
1
The effects of electrical stimulation of medial amygdala on the adrenocortical sensitivity to adrenocorticotrophin (ACTH) were investigated in hypophysectomized rats.
2
Subsequently, large fluctuations occurred in the adrenocorticotrophin concentration in the plasma of foetal sheep which were consistent with the stimulation of adrenocorticotrophin secretion.
1
The cardiovascular, fluid, electrolyte and endocrine responses to both steroid treatments were similar to those with corticotrophin.
2
The stress peptide corticotrophin-releasing factor (CRF) contributes to relapse to drug and alcohol seeking following withdrawal, although the cellular actions are poorly understood.
1
Plasma cortisol and adrenocorticotropichormone responses were similar to those found to experimentally-induced pain in normal subjects, i.e.
2
Plasma adrenocorticotropichormone levels were significantly higher in the CRF patients, possibly indicative of an underlying hypothalamic-pituitary-adrenal axis abnormality.
3
The serum cortisol and adrenal steroids levels were always nearly undetectable, but the adrenocorticotropichormone levels were extremely high.
4
However, blood pressure, mean heart rate and the plasma adrenocorticotropichormone level showed no significant differences between the two groups.
5
We studied four patients with adrenocorticotropichormone (ACTH)-independent hypercortisolism due to bilateral massive enlargement of the adrenal glands.
1
In all cases, cortisol level increased three-fold after adrenocorticotrophichormone.
2
Plasma adrenocorticotrophichormone (ACTH) and cortisol levels were determined at serial time points.
3
These sb mice also had significantly greater increases in plasma adrenocorticotrophichormone than mice with other genotypes after a stressful stimulus.
4
Low cortisol levels are almost universal in infants after treatment with dexamethasone, but the adrenal gland remains responsive to adrenocorticotrophichormone stimulation.
5
This study was designed to investigate the effect of chronic exercise on exercise-induced changes in plasma concentrations of adrenocorticotrophichormone (ACTH) and corticosterone in rats.
1
The best imaging approach for localizing ectopic ACTH-secreting tumors is not defined.
2
The rise in pressure was significant after 8 h of ACTH infusion.
3
The increase in cortisol levels was preceded by a corresponding ACTH increase.
4
The transport mechanism for precursor ACTH thus appears to recognize other prohormones.
5
ACTH insensitivity results from a group of rare autosomal recessive genetic defects.
6
Serum cortisol concentrations and ACTH stimulation tests were used to detect CIRCI.
7
In addition, laboratory examination showed elevated plasma ACTH and serum cortisol levels.
8
The responses to all doses of ET-1 were similar during ACTH infusion.
9
Plasma GH concentration was decreased but plasma cortisol and ACTH remained unaffected.
10
However, many patients with ACTH resistance lack an identifiable genetic aetiology.
11
Results: A greater decline in ACTH was observed after cortisol injection in PTSD.
12
Possible mechanism of elevated ACTH in this disorder was briefly discussed.
13
This tumour type frequently contains peptide hormones of the ACTH family.
14
Cortisol levels remained elevated and ACTH increased during the 13-day period.
15
The ACTH response to CRF attenuated by repeated administration of CRF.
16
ACTH did not further enhance the maximal effect of AII on c-fos expression.