Urinary bladder cancer that has material basis in abnormally proliferating cells derives from epithelial cells.
1Studies on HER-2 expression in bladder carcinoma have shown heterogeneous results.
2Brachytherapy has a key role to play in the treatment of bladder carcinoma.
3Pathologic examination showed similar histologic and immunohistochemical characteristics as the primary bladder carcinoma.
4Purpose: We report the presentation of brain metastases from bladder carcinoma.
5Results: Brain metastases from bladder carcinoma were commonly accompanied by uncontrolled systemic metastases.
6Conclusions: Overall survival after brain metastasis development in patients with bladder carcinoma was poor.
7However, the mechanisms responsible for angiogenesis in urinary bladder carcinoma patients are not well defined.
8The in vitro proliferation of both bladder carcinoma cell lines was also inhibited by TNP-470.
9However, no ultrasonic enhancement of doxorubicin growth inhibition in these human bladder carcinoma cells was observed.
10The data indicated that ARCON could achieve a therapeutic gain in patients with advanced bladder carcinoma.
11Photochemotherapy of bladder carcinoma in situ still constitutes a complex treatment protocol reserved for specialized centres.
12Background: The clinical behavior of the tumor in patients with locally advanced bladder carcinoma is unpredictable.
13Conclusion: This study indicates thatΔNp63 gene down-expression can reduce the invasion of bladder carcinoma cells in vitro.
14Computed tomography showed diffuse, circumferential, irregular, and lobulated thickening of the bladder wall suggestive of urinary bladder carcinoma.
15These data identify distinct regions of loss on chromosome 4 potentially involved in the late progression of bladder carcinoma.
16Conclusions: These findings indicate that TD mutations in the FGFR3 gene do not cause disease progression of bladder carcinoma.