Urinary bladder cancer that has material basis in abnormally proliferating cells derives from epithelial cells.
1 Studies on HER-2 expression in bladder carcinoma have shown heterogeneous results.
2 Brachytherapy has a key role to play in the treatment of bladder carcinoma .
3 Pathologic examination showed similar histologic and immunohistochemical characteristics as the primary bladder carcinoma .
4 Purpose: We report the presentation of brain metastases from bladder carcinoma .
5 Results: Brain metastases from bladder carcinoma were commonly accompanied by uncontrolled systemic metastases.
6 Conclusions: Overall survival after brain metastasis development in patients with bladder carcinoma was poor.
7 However, the mechanisms responsible for angiogenesis in urinary bladder carcinoma patients are not well defined.
8 The in vitro proliferation of both bladder carcinoma cell lines was also inhibited by TNP-470.
9 However, no ultrasonic enhancement of doxorubicin growth inhibition in these human bladder carcinoma cells was observed.
10 The data indicated that ARCON could achieve a therapeutic gain in patients with advanced bladder carcinoma .
11 Photochemotherapy of bladder carcinoma in situ still constitutes a complex treatment protocol reserved for specialized centres.
12 Background: The clinical behavior of the tumor in patients with locally advanced bladder carcinoma is unpredictable.
13 Conclusion: This study indicates thatΔNp63 gene down-expression can reduce the invasion of bladder carcinoma cells in vitro.
14 Computed tomography showed diffuse, circumferential, irregular, and lobulated thickening of the bladder wall suggestive of urinary bladder carcinoma .
15 These data identify distinct regions of loss on chromosome 4 potentially involved in the late progression of bladder carcinoma .
16 Conclusions: These findings indicate that TD mutations in the FGFR3 gene do not cause disease progression of bladder carcinoma .
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