The role of TLR4 in brainischemia has not been examined yet.
2
Enhanced glucose uptake protects cells during energy depletion including brainischemia.
3
Summary: Reversible brainischemia is a harbinger for subsequent ischemic stroke.
4
Four-vessel occlusion model of transient global brainischemia was used.
5
PGA1 treatment also significantly ameliorated motor dysfunction after brainischemia.
6
Here we report that PHD1 deficiency provides neuroprotection in a murine model of permanent brainischemia.
7
Masitinib reduced significantly brainischemia induced by experimental stroke and potentiated the therapeutic effect of rt-PA.
8
Background: Cognitive dysfunction and delirium after ICU are frequent and may partially result from brainischemia episodes.
9
These studies represent evidence that changes in the HPA axis play an important role in brainischemia.
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Neuronal injury from even brief periods of global brainischemia seems to be associated with deteriorating neurocognitive function.
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Taken together, our results suggest that PAFR activation might be crucial for the global brainischemia and reperfusion injury.
12
Objective: To review the pathophysiology of acute brainischemia and infarction and the evidence supporting various stroke reperfusion treatments.
13
The pathogenesis of acute brainischemia (ABI) is highly complex and involves multiple mechanisms including free radical generation.
14
Heat shock proteins (HSPs) induced by brainischemia may play an important role in neuroprotection from neuronal degeneration.
15
Mobilization after heart or brainischemia is well established, but it is unclear if this occurs after intestinal ischemia-reperfusion injury.
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Edaravone is the active substance of a Japanese medicine, which aids neurological recovery following acute brainischemia and subsequent cerebral infarction.