Significados de brain ischemia en inglés

The role of TLR4 in brain ischemia has not been examined yet.

Enhanced glucose uptake protects cells during energy depletion including brain ischemia.

Summary: Reversible brain ischemia is a harbinger for subsequent ischemic stroke.

Four-vessel occlusion model of transient global brain ischemia was used.

PGA1 treatment also significantly ameliorated motor dysfunction after brain ischemia.

Here we report that PHD1 deficiency provides neuroprotection in a murine model of permanent brain ischemia.

Masitinib reduced significantly brain ischemia induced by experimental stroke and potentiated the therapeutic effect of rt-PA.

Background: Cognitive dysfunction and delirium after ICU are frequent and may partially result from brain ischemia episodes.

These studies represent evidence that changes in the HPA axis play an important role in brain ischemia.

Neuronal injury from even brief periods of global brain ischemia seems to be associated with deteriorating neurocognitive function.

Taken together, our results suggest that PAFR activation might be crucial for the global brain ischemia and reperfusion injury.

Objective: To review the pathophysiology of acute brain ischemia and infarction and the evidence supporting various stroke reperfusion treatments.

The pathogenesis of acute brain ischemia (ABI) is highly complex and involves multiple mechanisms including free radical generation.

Heat shock proteins (HSPs) induced by brain ischemia may play an important role in neuroprotection from neuronal degeneration.

Mobilization after heart or brain ischemia is well established, but it is unclear if this occurs after intestinal ischemia-reperfusion injury.

Edaravone is the active substance of a Japanese medicine, which aids neurological recovery following acute brain ischemia and subsequent cerebral infarction.