1Normal immunoglobulin G did not have such effects on retinal cells.
2These nonspecific responses were transient and of the immunoglobulin G isotype.
3Serum immunoglobulin G, A, and M were measured by end-point nephelometry.
4Intrathecally produced immunoglobulin G was resolved into sharply focused, straight and easily identifiable fractions.
5H. pylori infection was measured serologically by immunoglobulin G antibody titers against H. pylori.
6Another patient had a below normal immunoglobulin G level.
7BBB permeability was detected with sodium fluorescein extravasation and further confirmed by immunoglobulin G immunohistochemistry.
8Serum samples were analyzed for immunoglobulin G antibodies against H. pylori by enzyme-linked immunosorbent assay.
9One month after boosting at age 4 years, menC immunoglobulin G and SBA levels increased significantly.
10The mechanisms involved in sequential immunoglobulin G (IgG) class switching are still largely unknown.
11The pH gradients were stable and the inter-gel reproducibilities of individual immunoglobulin G patterns were good.
12Conjugate consisted of immunoglobulin G fraction of goat antiserum against chicken IgG bound to horseradish peroxidase.
13The method was standardized by using human aggregated immunoglobulin G (IgG) to simulate immune complexes.
14The serum levels of antibodies for H. pylori immunoglobulin G were measured by using an ELISA test.
15Silver staining and nitrocellulose blotting with amplified immunoperoxidase detection of immunoglobulin G were used for protein visualization.
16Circulating immunoglobulin G antibody activity to the bundle-forming pilus subunit A was readily detected in all children.
Translations for immunoglobulin G