Normal immunoglobulin G did not have such effects on retinal cells.

These nonspecific responses were transient and of the immunoglobulin G isotype.

Serum immunoglobulin G, A, and M were measured by end-point nephelometry.

Intrathecally produced immunoglobulin G was resolved into sharply focused, straight and easily identifiable fractions.

H. pylori infection was measured serologically by immunoglobulin G antibody titers against H. pylori.

Another patient had a below normal immunoglobulin G level.

BBB permeability was detected with sodium fluorescein extravasation and further confirmed by immunoglobulin G immunohistochemistry.

Serum samples were analyzed for immunoglobulin G antibodies against H. pylori by enzyme-linked immunosorbent assay.

One month after boosting at age 4 years, menC immunoglobulin G and SBA levels increased significantly.

The mechanisms involved in sequential immunoglobulin G (IgG) class switching are still largely unknown.

The pH gradients were stable and the inter-gel reproducibilities of individual immunoglobulin G patterns were good.

Conjugate consisted of immunoglobulin G fraction of goat antiserum against chicken IgG bound to horseradish peroxidase.

The method was standardized by using human aggregated immunoglobulin G (IgG) to simulate immune complexes.

The serum levels of antibodies for H. pylori immunoglobulin G were measured by using an ELISA test.

Silver staining and nitrocellulose blotting with amplified immunoperoxidase detection of immunoglobulin G were used for protein visualization.

Circulating immunoglobulin G antibody activity to the bundle-forming pilus subunit A was readily detected in all children.