All patients were venesected within 24 h of admission to exclude an inflammatory state through evaluation of c-reactive protein.

Serum level of C-reactive protein, an inflammatory mediator, was also markedly increased.

In previous studies, the timing of C-reactive protein determination has varied widely.

There was no significant difference in circulating C-reactive protein or interleukin-6 concentrations.

Subclinical hypothyroidism was associated with higher concentrations of triglycerides and C-reactive protein.

Her low-density lipoprotein cholesterol and high-sensitivity C-reactive protein levels were not elevated.

PWV was also correlated with age, systolic blood pressure, and C-reactive protein.

C-reactive protein concentration and some blood coagulation profiles within the study period.

Vitamin E and fish oil supplements can help lower C-reactive protein.

PMID: 22467926 Similarly, no effect of C-reactive protein levels was observed.

Plasma C-reactive protein levels were measured and baseline coronary risk factors were collected.

Thus, C-reactive protein is unable to discriminate the causes of renal graft dysfunction.

Elevated C-reactive protein levels in patients with AF reflects an underlying inflammatory process.

Subsequently, the fever improved without antipyretics and her C-reactive protein level decreased rapidly.

RA was also significantly associated with type 2 DM and C-reactive protein levels.

Plasma C-reactive protein level should be considered as a prognostic factor in CVT.