1 There was a good correlation between 51Cr and lactate dehydrogenase release values.
2 KN-62 also antagonized the OA-enhanced release of lactate dehydrogenase from digitonin-permeabilized hepatocytes.
3 To assess cell toxicity, we measured cell viability and lactate dehydrogenase release.
4 Cell death was assessed using in situ end-labeling and lactate dehydrogenase release.
5 They also significantly correlated with serum soluble interleukin-2 receptor and lactate dehydrogenase levels.
6 Cultures were assayed for lactate dehydrogenase release and Trypan Blue uptake.
7 Cell viability was evaluated by propidium iodide and lactate dehydrogenase release.
8 Striking elevations of liver tests, particularly lactate dehydrogenase , were seen.
9 SiNPs induced a mild but dose-dependent increase of lactate dehydrogenase .
10 Diagnostic work-up revealed elevated gamma-glutamyl transferase and lactate dehydrogenase levels.
11 Cytotoxicity was evaluated by measuring 51Cr and lactate dehydrogenase release.
12 ELISA was used to measure the level of lactate dehydrogenase (LDH) in neurons.
13 Small, but not large platelets show a lactate dehydrogenase activity higher than that of controls.
14 Cytotoxic effects were determined using lactate dehydrogenase release assay.
15 All patients reported muscle weakness and had elevated muscle enzymes creatine kinase and lactate dehydrogenase .
16 Membrane integrity was analyzed by the lactate dehydrogenase assay.
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