1There was a good correlation between 51Cr and lactate dehydrogenase release values.
2KN-62 also antagonized the OA-enhanced release of lactate dehydrogenase from digitonin-permeabilized hepatocytes.
3To assess cell toxicity, we measured cell viability and lactate dehydrogenase release.
4Cell death was assessed using in situ end-labeling and lactate dehydrogenase release.
5They also significantly correlated with serum soluble interleukin-2 receptor and lactate dehydrogenase levels.
6Cultures were assayed for lactate dehydrogenase release and Trypan Blue uptake.
7Cell viability was evaluated by propidium iodide and lactate dehydrogenase release.
8Striking elevations of liver tests, particularly lactate dehydrogenase, were seen.
9SiNPs induced a mild but dose-dependent increase of lactate dehydrogenase.
10Diagnostic work-up revealed elevated gamma-glutamyl transferase and lactate dehydrogenase levels.
11Cytotoxicity was evaluated by measuring 51Cr and lactate dehydrogenase release.
12ELISA was used to measure the level of lactate dehydrogenase (LDH) in neurons.
13Small, but not large platelets show a lactate dehydrogenase activity higher than that of controls.
14Cytotoxic effects were determined using lactate dehydrogenase release assay.
15All patients reported muscle weakness and had elevated muscle enzymes creatine kinase and lactate dehydrogenase.
16Membrane integrity was analyzed by the lactate dehydrogenase assay.
Translations for lactate dehydrogenase