1
The development of autoimmunity as a result of target-related mutations is unique.
2
Patients have a risk of infections and autoimmunity associated with T lymphocytopenia.
3
Conclusions: Neural-specific autoantibodies are not uncommon in patients with ICI-related neurological autoimmunity.
4
Our findings may be evidence of associations between autoimmunity and psoriasis vulgaris.
5
There was no significant treatment-related toxicity and no clinical evidence of autoimmunity.
6
However, the mechanisms underlying the development of BALT during systemic autoimmunity remain unknown.
7
A new study validates the role of these common autoantigens in preventing autoimmunity.
8
However, both pathways led to tumor immunity and identical phenotypic manifestations of autoimmunity.
9
The link between infection and autoimmunity is not yet well understood.
10
For decades, research in autoimmunity has focused primarily on immune contributions to disease.
11
Signs of autoimmunity may be present, but no consistent pattern has been observed.
12
The presence of type B reactive T cells should be considered in autoimmunity.
13
Logistic regression analysis revealed that higher adiponectin levels were associated with thyroid autoimmunity.
14
The effect of adiponectin on thyroid autoimmunity will require more studies.
15
Abnormalities in these genes are likely involved also in lymphoid tumorigenesis and autoimmunity.
16
Mesenchymal stem cells have the capacity to maintain immune homeostasis and prevent autoimmunity.
