Significados de infarct myocardium en inglés

However, the participation of BMCs in the reconstruction of infarcted myocardium remains unclear.

Conclusion: Irrigated RF energy produced relatively large lesions in infarcted myocardium without thrombus formation.

However, limited data exist regarding the characteristics of irrigated RF lesions in infarcted myocardium.

Infarcted myocardium enhances during the first-pass MRI study unless there is microvascular or epicardial obstruction.

In infarcted myocardium, extracellular recordings exhibit multiple deflections due to irregular pathway of the electric impulse.

This study showed the different SI patterns between recently infarcted myocardium and chronic scar on the CE-cine MRI.

However, their in vivo reparative capability is limited due to lack of their survival in the infarcted myocardium.

In conclusion, myocardial imaging using thallium reinjection method may be useful for the assessment of viability of the infarcted myocardium.

Dynamic PET-CT imaging indicated rapid blood clearance via kidney filtration, and accumulation within a V β 3 -positive infarcted myocardium.

The SI of the infarcted myocardium on the CE-cine MRI was compared with that of the normal myocardium on the same image.

Conclusions: This study suggests that the MnDPDP-unenhanced area on the MR images includes not only the infarcted myocardium but also the stunned myocardium.

Objectives: The purpose of this study was to assess the effect of scar tissue composition on engraftment of progenitor cells into infarcted myocardium.

Furthermore, Hsp20-MSCs improved cardiac function of infarcted myocardium as compared with vector-MSCs, accompanied by reduction of fibrosis and increase in the vascular density.

Expansion (regional dilatation and thinning) of acutely infarcted myocardium in man has been shown to correlate with overall cardiac dilatation and rupture.

As a further control group, rats injected with autologous bone marrow mononuclear cells into the infarcted myocardium did not show increased susceptibility to PES.

However, the hUCB-MSCs showed remarkable variations in their therapeutic efficacy for repairing rat infarcted myocardium (including the process of angiogenesis) 8 weeks after transplantation.