Film school in the United Kingdom.
1NFTs, but not neuronal loss, were demonstrated within the central superior nucleus.
2A subset of the cases had NFTs in the medulla oblongata.
3Ventromedial temporal lobe NFTs probably represent the substrate for memory decline in MCI.
4Neuronal loss was not demonstrated within CSN, although NFTs were abundant within this nucleus.
5Numbers of NFTs were quantified in the same regions.
6The significance of the accumulation of NFTs for neuronal and cognitive function is still obscure.
7The NFTS's success is disproportionate to its size.
8It is possible that NFTs disrupt synaptic transmission and plasticity, leading to memory deficits and cognitive malfunction.
9These dentate granule cell NFTs could provide a proxy indicator of CARTS pathology in cases lacking substantial AD pathology.
10In this study we have analyzed and quantified the NFTs during external automatic defibrillation in 105 cardiac arrest patients.
11The NFTs were slightly more prominent than beta-amyloid peptide deposition in the progression from normal to MCI to EAD.
12Relative severity of neuronal loss or NFTs was usually consistent from level to level within nuclei; internuclear correlations were weaker.
13This uncertainty fuels doubts about the diagnostic importance of NFTs and NPs and leads to confusion regarding hypotheses of AD pathogenesis.
14In patients with MCI compared with controls, the only significant increase in NFTs in the neocortex was in the parietal lobe.
15Furthermore, this study will contribute to the discussion about the efficacy and specific and unspecific effects of NFTs in children with ADHD.
16Counts of NFTs within LC were also highest at mid level (p less than .05) in comparison with caudal level).