Film school in the United Kingdom.
1 NFTs , but not neuronal loss, were demonstrated within the central superior nucleus.
2 A subset of the cases had NFTs in the medulla oblongata.
3 Ventromedial temporal lobe NFTs probably represent the substrate for memory decline in MCI.
4 Neuronal loss was not demonstrated within CSN, although NFTs were abundant within this nucleus.
5 Numbers of NFTs were quantified in the same regions.
6 The significance of the accumulation of NFTs for neuronal and cognitive function is still obscure.
7 The NFTS 's success is disproportionate to its size.
8 It is possible that NFTs disrupt synaptic transmission and plasticity, leading to memory deficits and cognitive malfunction.
9 These dentate granule cell NFTs could provide a proxy indicator of CARTS pathology in cases lacking substantial AD pathology.
10 In this study we have analyzed and quantified the NFTs during external automatic defibrillation in 105 cardiac arrest patients.
11 The NFTs were slightly more prominent than beta-amyloid peptide deposition in the progression from normal to MCI to EAD.
12 Relative severity of neuronal loss or NFTs was usually consistent from level to level within nuclei; internuclear correlations were weaker.
13 This uncertainty fuels doubts about the diagnostic importance of NFTs and NPs and leads to confusion regarding hypotheses of AD pathogenesis.
14 In patients with MCI compared with controls, the only significant increase in NFTs in the neocortex was in the parietal lobe.
15 Furthermore, this study will contribute to the discussion about the efficacy and specific and unspecific effects of NFTs in children with ADHD.
16 Counts of NFTs within LC were also highest at mid level (p less than .05) in comparison with caudal level).
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