However, the identity of most redox-regulated mitochondrial proteins remains to be defined.

This process obviates the need for exogenous enzymes and redox mediator supplements.

These findings reveal a new layer of nitroso-redox imbalance in dystrophic cardiomyopathy.

These single-domain flavoenzymes display redox potentials compatible with electron transfer between partners.

Hence, there is interest in redox and metabolic modulators as anticancer agents.

In addition, the redox properties of the Te functionalities have been explored.

Blood free radical concentration was reduced, that is, CV improved redox homeostasis.

Manipulation of redox signaling may be beneficial in cancer patients receiving trastuzumab.

Solution and solid state CV studies revealed the redox-active characteristics of the complexes.

The redox balance in the renal cell seems, therefore, of the utmost importance.

CSK therefore provides a redox regulatory mechanism that couples photosynthesis to gene expression.

These markers play important roles in redox regulations throughout the body.

Importantly, disruption of redox organization is a common basis for disease.

The redox-sensitive transcription factors AP-1 and NF-kappaB have been implicated in these effects.

These results define a novel redox-based mechanism to propagate Fas-dependent apoptosis.

This redox cycle may constitute the principal physiologic function of bilirubin.