AdhE- mutants of E. coli fail to ferment glucose and introduction of adhEMeta restored the growth of such mutants when grown under fermentative conditions.
2
The adh.2C2 and adh.6D4 clones thus provide an accessible system for defining mechanisms controlling developmental plasticity in early T-cell development.
3
However, cloned adhEMeta did not confer ethanol utilization ability to Escherichia coli or to Pseudomonas aeruginosa, even though it was transcribed in both these hosts.
4
Conclusion: We conclude that rare ADH variants are specific for alcohol dependence.
5
Twenty-four hours after LDN, mean ADH levels had returned to normal values.
1
Copeptin is a sensitive and more stable surrogate marker for argininevasopressin.
2
The serum argininevasopressin level was low for the serum osmolality.
3
In this study, argininevasopressin hypothalamic gene expression was studied in cirrhotic rats.
4
The main cause of hyponatremia is nonosmotic secretion of argininevasopressin with resultant electrolyte-free water retention.
5
We report here the cloning of a complementary DNA encoding the hepatic V1a argininevasopressin receptor.
1
Patients who lack this antidiuretichormone suffer from central diabetes insipidus.
2
A single E2078 administration reduced plasma antidiuretichormone dose-dependently.
3
The syndrome of inappropriate secretion of antidiuretichormone seems to us to be a frequent complication of severe craniocerebral trauma.
4
Surgical intervention, in the case of acute craniocerebral trauma, does not result in a higher frequency of inappropriate secretion of antidiuretichormone.
5
The hyponatremia caused by syndrome of inappropriate secretion of antidiuretichormone (SIADH) was treated with normal saline and water restriction.
Uso de avp em inglês
1
However, the stimuli to AVP is likely different between cycling and running.
2
Results: Twenty-two patients were found to carry a pathogenic AVP-NPII gene mutation.
3
One AVP patient developed acute coronary syndrome with dose-dependent ECG changes.
4
No differences were found for non-social working memory or for AVP vs placebo.
5
We assessed the results of vascular embolization obtained using the AVP.
6
The magnitude of the AVP-induced vasoconstriction was significantly reduced by calphostin-C.
7
The modest decrease in PV was not the primary non-osmotic stimulus to AVP.
8
The contents of SS and AVP in hippocampus tissue were measured by radioimmunoassay.
9
Conclusions: Peripheral embolization with the AVP 4 was successful in the majority of patients.
10
At the maximum effect, pinacidil completely relaxed vasoconstriction in the continuing exposure to AVP.
11
These results suggest that periventricular dopaminergic mechanisms may act to inhibit hemorrhage-induced AVP secretion.
12
The copeptin assay may be a useful alternative to direct measurement of AVP concentration.
13
This study evaluated the AVP 4 in peripheral vascular embolization.
14
Measurement of AVP levels has limitations due to its short half-life and cumbersome detection method.
15
In ICU patients, AVP decreased to moderately elevated levels within 24 h after ED admission.
16
It has been proposed that this is because AVP facilitates baroreflex control of the circulation.