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PURPOSE-Evaluateclinical and laboratory aspects of the hepatocarcinoma treated in our service.
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Above results suggest a negative relationship between GSN expression and hepatocarcinoma cell apoptosis.
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A new nodule of hepatocarcinoma reappeared twelve months after surgery and a liver resection was performed.
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The present study reveals that cattle bile may be a potential alternative to bear bile for hepatocarcinoma therapy.
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In current study, we demonstrate the roles of GSN on anti-apoptosis of hepatocarcinoma cells by transcriptome RNA-seq method.
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Application of these scaffolds for tissue engineering was tested by encapsulation of hepatocarcinoma cell line (HepG2).
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CONCLUSION-Thehepatocarcinoma is rare in our service, eighty percent had advanced disease and 42% were positive for HBsAg.
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Background: Hepatitis C infection induces an acute and chronic liver inflammation that may lead to cirrhosis, liver failure, or hepatocarcinoma.
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Similarly, we observed decreased DNA fragmentation in JUNV-infected human hepatocarcinoma cells deficient for RIG-I when compared with that of RIG-I-competent cells.
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Thirty three patients with hepatocarcinoma were treated between 1989 and 1997, 23 were treated surgically or by chemotherapy.
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Similar inhibitory effects were observed when the GFP-HepG2 hepatocarcinoma cells treated with C6-8-conjugated CDots were implanted in nude mice.
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In human hepatocarcinoma and prostate cancer cells, the activation of ORs by odors modulates elementary physiological processes and leads to an inhibitory effect on proliferation.