In nine cases the hepatoma was discovered during emergency treatment for complications.
2
Human HepG2 hepatoma cells express a high number of insulin receptors.
3
In this study, we investigated FasL expression in 13 human hepatoma cell lines.
4
Expression of pattern recognition receptors was reconstituted in hepatoma cells by lentiviral transduction.
5
In contrast, Fu5AH rat hepatoma cells did not discriminate this compound from cholesterol.
1
Presentation of case: A 74-year-old man was diagnosed with advanced hepatocellularcarcinoma.
2
Thus, growth rate and vascularity in well-differentiated hepatocellularcarcinoma are closely correlated.
3
Methods: One hundred fifty-nine patients with recurrent hepatocellularcarcinoma were studied retrospectively.
4
Background: There is no effective systemic therapy for patients with hepatocellularcarcinoma.
5
Local recurrence is observed more frequently for metastases than for hepatocellularcarcinoma.
Uso de hepatocarcinoma em inglês
1
PURPOSE-Evaluateclinical and laboratory aspects of the hepatocarcinoma treated in our service.
2
Above results suggest a negative relationship between GSN expression and hepatocarcinoma cell apoptosis.
3
A new nodule of hepatocarcinoma reappeared twelve months after surgery and a liver resection was performed.
4
The present study reveals that cattle bile may be a potential alternative to bear bile for hepatocarcinoma therapy.
5
In current study, we demonstrate the roles of GSN on anti-apoptosis of hepatocarcinoma cells by transcriptome RNA-seq method.
6
Application of these scaffolds for tissue engineering was tested by encapsulation of hepatocarcinoma cell line (HepG2).
7
CONCLUSION-Thehepatocarcinoma is rare in our service, eighty percent had advanced disease and 42% were positive for HBsAg.
8
Background: Hepatitis C infection induces an acute and chronic liver inflammation that may lead to cirrhosis, liver failure, or hepatocarcinoma.
9
Similarly, we observed decreased DNA fragmentation in JUNV-infected human hepatocarcinoma cells deficient for RIG-I when compared with that of RIG-I-competent cells.
10
Thirty three patients with hepatocarcinoma were treated between 1989 and 1997, 23 were treated surgically or by chemotherapy.
11
Similar inhibitory effects were observed when the GFP-HepG2 hepatocarcinoma cells treated with C6-8-conjugated CDots were implanted in nude mice.
12
In human hepatocarcinoma and prostate cancer cells, the activation of ORs by odors modulates elementary physiological processes and leads to an inhibitory effect on proliferation.