Conclusions: Inherited thrombophilia is associated with an increased risk of upper-extremity DVT.
2
Additional considerations regarding HT choice include thrombotic risk and disorders associated with thrombophilia.
3
Objective: To determine to what extent adverse pregnancy outcomes are associated with thrombophilia.
4
Thus, at present, universal screening for thrombophilia in pregnancy cannot be justified clinically.
5
Oral contraceptives increase the risk only when combined with inherited thrombophilia.
1
These changes demonstrate a possible mechanism for hypercoagulability during adult VA ECMO.
2
Searching for primary malignancy should include HCC as frequent cause of hypercoagulability.
3
COVID-19 is also associated with an increased risk of hypercoagulability and venous thromboembolism.
4
Clot rate could emerge as a potential predictor of hypercoagulability in these patients.
5
Numerous new factors associated with hypercoagulability have been described in the past few years.
1
In two cases, an ultrasound examination suggested a diagnosis of venousthrombosis.
2
Objective: Data regarding cerebral venousthrombosis in North Africa are scarce.
3
Background: Hyperhomocysteinemia is an independent risk factor for atherothrombotic disease and venousthrombosis.
4
We report the case of an unusual and early neonatal renal venousthrombosis.
5
The risk for both arterial and venousthrombosis increases with age.
1
Underlying prothromboticstate and absence of anticoagulant therapy were independent predictors for thrombosis.
2
We also found changes in biomarkers showing evidence that rhEPO induced a prothromboticstate.
3
The effect of vasodilating beta-blockers on endothelial function and prothromboticstate has not been investigated.
4
The role of platelets in the development of atherosclerosis and obesity-related prothromboticstate is still under investigation.
5
This review summarizes the cellular and molecular mechanisms by which adherent leukocytes can induce a prothromboticstate.
1
Diabetes mellitus (DM) is characterized by a hypercoagulabilitystate and many of these disorders are corrected with adequate metabolic control.
1
There was no association between venousthromboembolism and plasma prothrombin antigen level.
2
Only three of the 515 patients had symptomatic venousthromboembolism on follow-up.
3
No patient in the matched cohort had a venousthromboembolism or seizure.
4
Background: Systemic inflammatory diseases have been associated with increased risk of venousthromboembolism.
5
Patients with deep-vein thrombosis require long-term anticoagulant treatment to prevent recurrent venousthromboembolism.
Uso de hypercoagulable em inglês
1
Women with a past medical history of P-EC have a baseline hypercoagulable state postpregnancy.
2
Congenital and acquired hypercoagulable states arise from an imbalance between procoagulant and anticoagulant forces.
3
This includes a hypercoagulable state and the associated thromboembolism.
4
Thus, there is ongoing dispute about the impact of a hypercoagulable state upon progression of atherosclerosis.
5
Our findings suggest that a hypercoagulable state in heart failure can be modified by warfarin therapy.
6
Heart failure is associated with a hypercoagulable state.
7
Blood workup for preexisting hypercoagulable state was normal.
8
More recently, the hypercoagulable state which results from diminished response to APC has also been associated with arterial thrombosis.
9
Increased survival in recent years is consistent with improved management of hypercoagulable states as well as improved general care.
10
Thrombocytosis was shown to affect the blood hypercoagulable state and also have a negative prognostic value for GC patients.
11
Conclusions: An increased platelet aggregability contributes to the hypercoagulable states, that may increase the incidence of thrombosis in such patients.
12
The risk increases in patients with central lines and hypercoagulable states such as with malignancy, thrombophilia, or use of oral contraceptive pills.
13
The observed association likely does not reflect cause and effect, but rather that low serum albumin reflects a hyperinflammatory or hypercoagulable state.
14
A single-center, randomized, double-blind, placebo-controlled trial was performed to test the hypothesis that warfarin will modify a hypercoagulable state in heart failure.
15
It has been estimated that up to 10% of hypercoagulable state manifestations in patients with inflammatory bowel disease (IBD) are ischemic strokes.
16
We conclude from this pilot study that femorotibial PTA produces a hypercoagulable state which may result in failure of early patency due to rethrombosis.