In contrast, lipid A was a potent inducer of the PMNL response.
2
The studies reported here were undertaken to determine whether influenza virus could replicate within PMNL.
3
Both host- and virus-specific proteins were produced within PMNL.
4
Conclusion: Activated PMNLs enhance production of leukocyte MPs with increased adhesion molecules in patients with sepsis.
5
This study analyzed the ability of mucosally administered bacterial components to activate IL-6 and PMNL responses.
6
A significant correlation was found between the number of infected PMNLs and the number of infected HELF cells.
7
However, we also found interactions between the effects of haplotype and biopsy for body temperature, heart rate, and PMNL.
8
Negative PCR results for PMNL after antiviral therapy indicate recovery, and fewer unwanted positive results occur compared to PBMC and plasma.
9
By using indirect immunofluorescence techniques combined with flow cytometry, the expression of newly synthesized viral antigens was detected in virus-infected PMNL.
10
Treatment was discontinued only after disappearance of IEA-positive PMNLs from blood (the last marker of infection to become negative).
11
In conclusion, the measurement of transport of 3OMG in PMNLs may be useful for the study of glucose transport in clinical investigations.
12
However, overall interpretation of the transcriptome data agreed in part with literature documenting a potentially detrimental, chronic activation of PMNL in response to overfeeding.
13
Plaque assays on supernatant fluid from infected PMNL showed that infectious progeny were not produced, indicating that influenza virus infection of PMNL is abortive.
14
Blood for PMNL isolation and metabolite analysis was collected at -14 and +7 days relative to parturition.