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In the present paper the fluorescence properties of C60-glucocorticoids were firstly investigated.
2
We investigated whether cosyntropin testing predicts treatment response to glucocorticoids in CAP.
3
All of the 5 patients in stage III accepted glucocorticoids after diagnosis.
4
At the same time, the use of glucocorticoids was also very important.
5
The response to traditional therapy such as glucocorticoids may also be compromised.
1
Iatrogenic osteoporosis is a very common secondary osteoporosis is found in patients treated with large dosage of glucocorticosteroid of long duration.
2
The efficacy of a glucocorticosteroid-inducible promoter was assessed in controlling transgene expression following lentivirus-mediated gene transfer to ovine and human corneas.
3
The duration of treatment of primary disease with glucocorticosteroid is 1 to 3.5 years, with the average of 1.56 years.
4
At presentation, all patients except one had been inappropriately treated with glucocorticosteroid (insufficiently dosed or tapered too fast) for longer than 2 weeks.
5
The mechanisms appear to involve effects at the cell membrane as well as nuclear actions mediated by intracellular mineralo- and glucocorticosteroid receptors (MR and GR).
Usage of glucocorticoid in English
1
Objective: To determine the effectiveness of glucocorticoid treatment in children with croup.
2
The results suggest that THS was stable following treatment with glucocorticoid therapy.
3
The magnitude of this reduction was similar with treatment of either glucocorticoid.
4
Only men without acute disease exacerbation or systemic glucocorticoid treatment were included.
5
FKBP5 regulates the cortisol-binding affinity and nuclear translocation of the glucocorticoid receptor.
6
We also examined serum bone turnover markers before and after glucocorticoid replacement.
7
Objectives: Patients receiving glucocorticoid treatment are prone to develop metabolic complications.
8
We suggest that the extent of glucocorticoid action partly explains these interindividual differences.
9
Cohen himself published some of the early work on glucocorticoid resistance.
10
The classification results agree with the antagonistic activities of glucocorticoid and mineralocorticoid receptors.
11
Conflicting data have been previously reported concerning glucocorticoid effects on eosinopoiesis.
12
Awareness of this possibility and the use of glucocorticoid stress coverage are advised.
13
Dysregulation of the stress response is associated with elevated glucocorticoid levels.
14
These results indicate a glucocorticoid regulation of CART in the arcuate.
15
Binding of HNF-6 to DNA is required for inhibition of glucocorticoid receptor activity.
16
The long-term impact of fetal exposure to mitotane and glucocorticoid requires further investigation.